Ankylosing Spondylitis: Diagnosis and Management by Barend J. van Royen, Ben A. C. Dijkmans

By Barend J. van Royen, Ben A. C. Dijkmans

The 1st and simply interdisciplinary advisor devoted to the topic, this reference leads readers throughout the pathogenic, genetic, medical, and biomechanical elements of ankylosing spondylitis (AS) and addresses concerns on the topic of results overview, clinical therapy, surgical problems, affliction administration, and genetics.

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Extra info for Ankylosing Spondylitis: Diagnosis and Management

Example text

Ankylosis tibia, fibula and tarsus bones. Source: From Ref. 51. of juvenile chronic arthritis), and arthritis associated with inflammatory bowel disease (56–58). In 1991, the European Spondyloarthropathy Study Group (ESSG) modified this disease grouping to accommodate undifferentiated forms of spondyloarthropathy (59). Among the many landmarks in the history of AS and its relationship to the other spondyloarthropathies, perhaps the most important were the revelations of an infectious etiology and a genetic predisposition to AS.

The principal function of class I molecules is the presentation of endogenous peptides to CD8þ T lymphocytes to induce protective immune responses. Class I molecules conventionally consist of polymorphic HLA heavy chains which form a heterodimer with b2m (54). The membrane distal domains of the HC form a peptide-binding groove, which consists of several strands of b-pleated sheets topped by two antiparallel walls of a helices. MHC class I molecules can bind many different peptides with restrictions with regard to the length and sequence of the peptide (55).

68. Mielants H, Veys EM, Joos R, Noens I, Cuvelier C, De Vos M. HLA antigens in seronegative spondylarthropathies. Reactive arthritis and arthritis in ankylosing spondylitis: relation to gut inflammation. J Rheumatol 1987; 14:466–467. 2 Epidemiology, Pathogenesis, and Genetics of Ankylosing Spondylitis Andrew E. Timms, B. Paul Wordsworth, and Matthew A. K. EPIDEMIOLOGY Ankylosing spondylitis (AS) is one of the most common inflammatory rheumatic diseases, but estimates of its prevalence vary considerably, even in similar ethnic groups.

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